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Inhibition of PGC-1 expression has been shown to result in decreased expression of the mitochondrial fusion protein Mfn2, increased expression of p-Drp1, and increased mitochondrial fragmentation (Wang et al., 2019d)

However, the absence of large-scale human clinical trials is a genuine limitation

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Truncated sequences, deletion analogs, and racemized byproducts act as molecular saboteurs, competing for receptor binding sites, generating false signals in cell-based assays, and rendering months of research irreproducible